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Consistent with that hypothesis and based on the antioxidant effects of NRF2 activation in other systems [9,21,22], we believe that the most likely mechanism for the restoration of NO sensitivity by Protandim in the arterioles of HS-fed hamsters is maintenance of soluble guanylyl cyclase function via reduction of vascular oxidative stress

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Glucose-independent glutamine metabolism via TCA cycling for proliferation and survival in B cells